Sleep-apnoea

Excess weight is the primary modifiable risk factor for snoring and obstructive sleep apnoea. Weight loss reduces pharyngeal fat, increases upper airway calibre, and substantially reduces AHI. This article reviews the mechanism, the evidence on dose-response, and when weight loss alone is sufficient versus when CPAP is required.

Excess adipose tissue around the pharynx directly narrows the upper airway, increasing both snoring and obstructive sleep apnoea (OSA) severity. Weight loss has strong evidence for reducing OSA severity and snoring — but effect size depends on baseline weight, degree of weight loss achieved, and anatomical factors. This article covers the mechanisms, evidence, and realistic outcomes.

Excess weight is the primary modifiable cause of obstructive snoring and sleep apnoea. Weight loss reduces neck circumference and parapharyngeal fat, directly improving airway patency. Here is the clinical evidence and practical approach.

Obstructive sleep apnoea is strongly associated with excess weight through mechanical upper airway narrowing. Weight loss produces dose-dependent improvement in OSA severity and can achieve remission in mild-moderate cases. This article reviews the evidence.

Weight loss reduces snoring through reduction of pharyngeal fat and improved upper airway calibre. The evidence on dose-response, the distinction between simple snoring and OSA, and the NHS assessment pathway are reviewed here.